Quotes about essay writing
Tuesday, August 25, 2020
Population growth and natural resource availability Research Paper
Populace development and characteristic asset accessibility - Research Paper Example The mindfulness with respect to the need of the creation framework is expanding step by step so as to fuse the objectives of maintainable turn of events. For dealing with this it is significant that the regular assets are inexhaustible, as they are viewed as the markers for maintainability of the monetary elements. This methodology demonstrates that the normal assets are viewed as a connection among condition and social orders. The later subject in conversation identified with assets and populace development is viewed as the importance of the nature of condition which is estimated by the nonattendance of water and air contamination and the supply of woods in a specific district. As indicated by this view, condition may not be considered as a factor which restricts the profitability with the development of populace yet it would prefer to be characterized as factor whose quality is debased with the expansion in populace. The extending populace expands the requirement for an arable land; it is a fundamental driver behind deforestation and furthermore supports the change of woodlands into land for utilizing it for various purposes. It is additionally viewed as the significant reason for air, water and land contamination. On the opposite side ecological debasement may force an impact on the characteristic assets diminishing the minor result of the work through advancing lower earnings (Hinrichsen and Robey). Before, the disturbing circumstance was viewed as the reduction of common assets. There are numerous researchers who have been computing that term till which the stores of oil and coal will last. Along these lines at present the association among man and common habitat is viewed as a wellspring of caution. In each characteristic environment, the expansion in populace forces an incredible weight on it. This weight turns out to be progressively huge if there should arise an occurrence of the human populace, as this expansion isn't simply numerical yet additionally brings along the enhancement and formation of new needs. This subjective
Saturday, August 22, 2020
Web editing essays
Web altering articles One of the significant errands of the article staff is, the catch of the client wishes gives by the Key record director, media-reasonable verbally to move. This contains material quest and content change for the particular needs of a sight and sound item. By a totally unique conduct of use of the clients in examination with haptic distributions like books, magazines and papers the editorial manager must comprehend a colossal number of Guidelines to be dynamic effectively in the particular circumstance sight and sound. Individuals don't peruse sites a similar way they read print material. There are three common key attributes that influence how web clients respond to online substance: Web clients are dynamic, not uninvolved like book perusers The more extended the content, the more outlandish they are to understand it The client need to find solutions to his inquiries rapidly Great duplicate is fundamental to guaranteeing that the focused on peruser crowd and the clients can see how the site functions and what advantage it holds for them. 1.2. Tasks of the sight and sound branch 4 2.1.2. Separation of content and context 7 2.2. Book culture versus Web culture 7 4.2. Principles of copywriting for the web 14 The mixed media branch when all is said in done isn't anything but difficult to class, on the grounds that the entire branch is quick and the single business fields are fanned out comprehensively and change over the span of the time. In any case, the idea Multimedia encases in the limited sense the accompanying principle business fields: E showcasing, Internet, Intranet, Extranet, benefits in the B2B commercial centers, web existences, Content administration frameworks, Customer Relationship the executives frameworks, E-Commerce and web based promoting exercises. Mixed media applications must be all the more in an unexpected way (content, photograph and realistic expressions) and all the more powerfully (sound, liveliness and video) than other media types, in light of the chance of the intelligent use. The undertakings of the sight and sound branch is comprised of the accompanying 4 ... <!
Friday, August 7, 2020
How Long Does TCP Stay in Your System
How Long Does TCP Stay in Your System Addiction Drug Use Prescription Medications Print How Long TCP Stays in Your System By Buddy T facebook twitter Buddy T is an anonymous writer and founding member of the Online Al-Anon Outreach Committee with decades of experience writing about alcoholism. Learn about our editorial policy Buddy T Medically reviewed by Medically reviewed by Steven Gans, MD on December 17, 2015 Steven Gans, MD is board-certified in psychiatry and is an active supervisor, teacher, and mentor at Massachusetts General Hospital. Learn about our Medical Review Board Steven Gans, MD Updated on July 29, 2019 Andrew Brookes/Getty Images More in Addiction Drug Use Prescription Medications Cocaine Heroin Marijuana Meth Ecstasy/MDMA Hallucinogens Opioids Alcohol Use Addictive Behaviors Nicotine Use Coping and Recovery Estimating how long TCP (Tenocyclidine) is detectable in the body depends on many variables, including which kind drug test is being used. TCPâ"also known as N-[1-(2-thienyl)cyclohexyl] piperidineâ"can be detected for a shorter time with some tests but can be visible for up to three months in other tests. The timetable for detecting TCP in the system is also dependent upon each individuals metabolism, body mass, age, hydration level, physical activity, health conditions, and other factors, making it almost impossible to determine an exact time TCP will show up on a drug test. The following is an estimated range of times, or detection windows, during which TCP can be detected by various testing methods: Urine: TCP can be detected in the urine for 2-5 days.Blood: A blood test can detect TCP for up to 24 hours.Saliva: A saliva test can detect TCP for up to 1-5 daysHair: TCP, like many other drugs, can be detected with a hair follicle drug test for up to 90 days. Preventing an Overdose TCP is an analog of phencyclidine (PCP) in which the phenyl substituent is replaced with a thiophene group. It is intended for forensic and research applications only. However, when it is abused, TCP produces effects similar to the dissociative drug PCP except perhaps even more intense. The drug was placed on the list of Schedule 1 controlled substances when it was being abused in the 1970s and 1980s, but it is rarely used today. A TCP overdose can produce symptoms similar to a PCP overdose, which include: Low blood pressure and pulse rateSlow breathingNauseaVomitingBlurred visionDizziness If if you think someone has taken an overdose of TCP, call 9-1-1 immediately or the poison control center at 1-800-222-1222. TCP Is a Dangerous Chemical Coming in contact with Tenocyclidine itself can be harmful. It can cause skin irritation and serious eye damage. TCP can also cause mucous membrane and upper respiratory tract irritation. According to the manufacturers Safety Data Sheet, if someone inhales Tenocyclidine the should be moved into a fresh air environment immediately and administered oxygen. In the case of skin contact with Tenocyclidine, the area should be washed with soap and water for at least 15 minutes and any contaminated clothing removed. Medical attention should be sought immediately, according to the safety precautions. If you get Tenocyclidine in your eyes, you should hold your eyelids apart and flush eyes with water for at least 15 minutes. Your eyes should be examined and tested by a trained professional, the drugs manufacturer recommends.
Saturday, May 23, 2020
Shakespearean Insults From A to Z
William Shakespeare is one of the best insult-slinging writers in the English language. Do you ever find yourself wishing you had an inventive way to let off steam? Try some of these clever Shakespearean quips, organized alphabetically by the work in which they are found. Shakespearean Insults Alls Well that Ends Well (2.3.262)ââ¬Å"You are not worth another word, else Id call you knave.â⬠As You Like It (3.2.248)ââ¬Å"I do desire we may be better strangers.â⬠The Comedy of Errors (4.2.22-5)ââ¬Å"He is deformed, crooked, old and sere, /à Ill-faced, worse bodied, shapeless everywhere; /à Vicious, ungentle, foolish, blunt, unkind; /à Stigmatical in making, worse in mind.â⬠The Comedy of Errors (4.4.24)ââ¬Å"Thou whoreson, senseless villain!â⬠Coriolanus (2.1.36)ââ¬Å"You abilities are too infant-like for doing much alone.â⬠Coriolanus (2.1.59)ââ¬Å"They lie deadly that tell you you have good faces .â⬠Coriolanus (2.1.91)ââ¬Å"More of your conversation would infect my brain.â⬠Coriolanus (5.1.108-9)ââ¬Å"For such things as you, I can scarce think theres any, yere so slight.â⬠Coriolanus (5.4.18)ââ¬Å"The tartness of his face sours ripe grapes.â⬠Cymbeline (1.1.128)ââ¬Å"Away! Thourt poison to my blood.â⬠Hamlet (2.2.198) ââ¬Å"They have a plentiful lack of wit.â⬠Hamlet (5.2.335-6)ââ¬Å"Here, thou incestuous, murderous, damned Dane, /à Drink off this potion!â⬠1 Henry IV (2.4.225-6)ââ¬Å"This sanguine coward, this bed-presser, this horseback-breaker, this huge hill of flesh!â⬠1 Henry IV (2.4.227-9)ââ¬Å"Sblood, you starveling, you elf-skin, you dried neats tongue, you bulls pizzle, you stock-fish! O for breath to utter what is like thee! you tailors-yard, you sheath, you bowcase; you vile standing-tuck!â⬠1 Henry IV (3.3.40)ââ¬Å"Thereââ¬â¢s no more faith in thee than in a stewed prune.â⬠2 Henry IV (2.4.120-22)ââ¬Å"Away, you cut-purse rascal! you filthy bung, away! By this wine, Ill thrust my knife in your mouldy chaps, an you play the saucy cuttle with me. Away, you bottle-ale rascal! you basket-hilt stale juggler, you!Henry V (2.1.100)ââ¬Å"O braggart vile and damned furious wight!â⬠Henry V (3.2.30)ââ¬Å"He is white-livered and red-faced.â⬠1 Henry VI ( 3.2.54)ââ¬Å"Hag of all despite!ââ¬Å"1 Henry VI (5.4.30-1)ââ¬Å"Take her away; for she hath lived too long, /à To fill the world with vicious qualities.â⬠3 Henry VI (5.6.54-5)ââ¬Å"Teeth hadst thou in thy head when thou wast born, /à To signify thou camest to bite the world.â⬠Julius Caesar (1.1.36)ââ¬Å"You blocks, you stones, you worse than senseless things!â⬠King Lear (2.2.14-24)ââ¬Å"A knave; a rascal; an eater of broken meats; base, proud, shallow, beggarly, three-suited, hundred-pound, filthy, worsted-stocking knave; a lily-livered, action-taking knave, a whoreson, glass-gazing, super-serviceable finical rogue; one-trunk-inheriting slave; one that wouldst be a bawd, in way of good service, and art nothing but the composition of a knave, beggar, coward, pandar, and the son and heir of a mongrel bitch: one whom I will beat into clamorous whining, if thou deniest the least syllable of thy addition.â⬠King John (4.3.105)ââ¬Å"O you beast! /à Ill so maul you and your toasting-iron, /à That you shall think the devil is come from hell.â⬠Measure for Measure (2.1.113)ââ¬Å"You are a tedious fool.â⬠Measure for Measure (3.1.151-3)ââ¬Å"O faithless coward! O dishonest wretch! /à Wilt thou be made a man out of my vice?â⬠Measure for Measure (3.2.56)ââ¬Å"Some report a sea-maid spawnââ¬â¢d him; some that he was begot between two stock-fishes. But it is certain that when he makes water his urine is congealed ice.â⬠The Merry Wives of Windsor (2.3.21)ââ¬Å"Thou art a Castilian King urinal!â⬠The Merry Wives of Windsor (5.5.60)ââ¬Å"Vile worm, thou wast oââ¬â¢erlookââ¬â¢d even in thy birth.â⬠Othello (4.2.50)ââ¬Å"Heaven truly knows that thou art false as hell.â⬠Pericles (4.6.156)ââ¬Å"Thy food is such /à As hath been belchd on by infected lungs.â⬠Richard III (1.2.58)ââ¬Å"Thou lump of foul deformity!â⬠Richard III (1.2.159)ââ¬Å"Out of my sight! thou dost infect my eyes.â⬠The Taming of the Shrew (4.1.116)ââ¬Å"You peasant swain! You whoreson malt-horse drudge!â⬠The Tempest (3.2.29-30)ââ¬Å"Why, thou deboshed fish thou...Wilt thou tell a monstrous lie, being but half a fish and half a monster?â⬠Troilus and Cressida (2.1.10)ââ¬Å"Thou bitch-wolfs son!â⬠Troilus and Cressida (2.1.16-7)ââ¬Å"I think thy horse will sooner con an oration than /à thou learn a prayer without book.â⬠Troilus and Cressida (2.1.41)ââ¬Å"Thou sodden-witted lord! thou hast no more brain than I have in mine elbows.â⬠Troilus and Cressida (4.2.31)ââ¬Å"Go hang yourself, you naughty mocking uncle!â⬠Troilus and Cressida (2.1.106)ââ¬Å"I shall cut out your tongue.â⬠/à ââ¬Å"Tis no matter, I shall speak as much wit as thou afterwards.ââ¬
Tuesday, May 12, 2020
The Sword in the Stone Disneys Version vs. T.H. White
The story of King Arthur is widely known, either his beginnings told in The Sword in the Stone or how he led the Knights of the Round Table. While there are many version of his story T. H. Whiteââ¬â¢s written version and Disneyââ¬â¢s animated version of The Sword in the Stone are two of the most recognized versions. Most movies have the ability to embody the original intent of the book they were based upon. Disneyââ¬â¢s movie version of T. H. Whiteââ¬â¢s rendition of The Sword in the Stone, however, while portraying the correct story, does not truly convey enough elements of Whiteââ¬â¢s version to be effective in telling the original story. The characterization and Merlynââ¬â¢s ââ¬Ëlessonsââ¬â¢ within the movie inhibit the film from being an effective portrayal of theâ⬠¦show more contentâ⬠¦H. Whiteââ¬â¢s novel. The most important character in the story of King Arthur in both versions is Arthur himself or ââ¬Å"Wartâ⬠as he was known as before becoming King. While the movie does a decent job at portraying him as this weak unaware character, the movie does not give him enough credit for the feats he accomplishes himself. In the film, when Wart is transformed into a fish, Archimedes ultimately has to save Wart from the pike that almost kills him. However, in the book Wart manages to escape from the Pike by his own volition of knowledge, instead of the sheer power the Pike was using. Another time when Wart is portrayed as incompetent is when he is first introduced as a character. He ruins Kayââ¬â¢s shot at the deer and he is not even able to get the arrow back with ease. However as the story develops in the book, Wartââ¬â¢s character also develops. While he might start out as weak, he eventual grows into an intellectual character with depth and confidence. During his lesson with th e birds of prey and his ordeal, Wart is put into a situation where he needs to outsmart Colonel Cully. He does this through distracting the Colonel by using fear as a tool: ââ¬Å"There is a cat behind you,â⬠said the Wart calmly, ââ¬Å"or a pinemarten. Lookâ⬠(The Sword in the Stone). On the other hand, during his lesson with the geese Lyo-lyok helped him analyze the inhumane characteristics of humanity when she said: ââ¬Å"But what creature couldShow MoreRelatedStrategic Marketing Management337596 Words à |à 1351 PagesCompetitive strength â⬠¢ Market share â⬠¢ Relative share â⬠¢ Relative quality â⬠¢ Patents â⬠¢ Customer coverage Performance Value-added structure â⬠¢ Investment intensity â⬠¢ Fixed vs liquid assets â⬠¢ Capacity utilization â⬠¢ Productivity â⬠¢ Make vs buy People and organization â⬠¢ Lean organization â⬠¢ Participative culture â⬠¢ Incentives â⬠¢ Training â⬠¢ Insiders vs outsiders Figure 3.26 PIMS can quantify how strategic factors drive performance The second group describes how a business differs from its competitors in its
Wednesday, May 6, 2020
Richard III Power of Language and Own Villainy Free Essays
Deformed in body and twisted in mind, Richard is in every way the dominant character of the play, to the extent that he is both the playââ¬â¢s protagonist and major villain. He is selfish, evil, corrupt, sadistic, and manipulative. His intelligence, political brilliance, and dazzling use of language keeps the audience fascinated and his subjects and rivals under his control. We will write a custom essay sample on Richard III: Power of Language and Own Villainy or any similar topic only for you Order Now At the beginning of the play, it is made clear to the audience that Richard has no justification for seizing the throne. This is because England is obviously not oppressed or subject to tyranny as the lengthy civil war has just ended, and Richardââ¬â¢s oldest brother, King Edward IV, now sits on the throne. Richard himself, states that ââ¬ËAll the clouds that loured upon our houseââ¬â¢ (1. 1, 3), the house of York, has been dispelled by the ââ¬Ëson of Yorkââ¬â¢ (1. 1, 2), King Edward IV. However, Richard intends to upset the kingdom by seizing power for himself. He says that ââ¬Ësince I cannot prove a lover to entertain these fair well-spoken days, I am determined to prove a villainââ¬â¢ (1. , 28). This simply means that since Richard was not made to be a lover, he has no use for peace, and will happily destroy peace with his crimes. This shows Richardââ¬â¢s unabashed enjoyment of his own villainy as he can so blithely toss aside all of the things that the rest of humanity cherishes. Richard III is an intense exploration of the psychology of evil, and that exploration is centred on the workings of Richardââ¬â¢s mind and the methods he uses to manipulate, control, and injure others for his own gain. Perhaps more than any other play by Shakespeare, the audience of Richard III experiences a complex, indefinite, and highly erratic relationship with the main character. Richard is clearly a villain as he declares outright in his very first speech that he intends to stop at nothing to achieve his ultimate goal of becoming king. However, despite his open allegiance to evil, he has such a charismatic and fascinating personality that, for much of the play, we are likely to sympathize with him, or are at least impressed by him. In this way, our relationship with Richard reflects the other charactersââ¬â¢ relationships with him, conveying a powerful sense of the force of his personality. Even characters such as Lady Anne, who have an explicit knowledge of his wickedness, overlook his dishonesty and violent behaviour and allow themselves to be seduced by his brilliant wordplay, his skilful argumentation, and his relentless pursuit of his selfish desires. Richardââ¬â¢s long, fascinating soliloquys, in which he outlines his plans and gleefully confesses all his evil thoughts, are central to the audienceââ¬â¢s experience of Richard. Shakespeare uses these soliloquys brilliantly to control the audienceââ¬â¢s impression of Richard, enabling this manipulative protagonist to work his charm on the audience. In Act I, scene i, for example, Richard offers a pretext for his villainy towards others by pointing out that he is unloved, and that he is unloved because of his physical deformity. Richard himself is brutally honest about his appearance. He admits to being imperfectly shaped and blames premature birth for his condition. He knows that he is ââ¬Ënot shaped for sportive tricksââ¬â¢ (1. 1, 14) and while others delight in ââ¬Ëan amorous looking glassââ¬â¢ (1. , 14), his misshapen body creates a ââ¬Ëshadow in the sunââ¬â¢ (1. 1, 26) that alienates him from others. Hence, Lady Anne calls Richard a ââ¬Ëlump of foul deformityââ¬â¢ (1. 2, 57) in Act I, scene ii. This proves that Richardââ¬â¢s claim not only makes the other characters of the play seem like the villains for punishing him for his appearance, but also makes it easy for the audience to sympathize with Richard during the first scenes of the play and even hope that he will succeed despite his obvious villainy. It quickly becomes apparent, however, that Richard simply uses his deformity as a tool to gain the sympathy of others, including the audience. This is already noticeable in his very first speech as Richard seems to take a deliberate perverse delight in his outward shape. He chooses words such as, ââ¬Ëcheatedââ¬â¢, ââ¬Ëdeformedââ¬â¢, ââ¬Ëunfinishedââ¬â¢, ââ¬Ëhalf made upââ¬â¢, ââ¬Ëdogs barkââ¬â¢ at him as he passes by because of his ââ¬Ëdeformityââ¬â¢ to describe himself. Richardââ¬â¢s unabashed villainy is a much more natural part of his character than simple bitterness about his ugly body. Nevertheless, he still manages to use speech to win our trust, and he repeats this throughout his struggle to be crowned king. An interesting secondary theme of Richard III is the power of language, or the importance of language in achieving political power. Language may not always be a necessary instrument of power, but for Richard, it is a crucial weapon. As we have seen, it is with his extraordinary skills with words that allows him to ridicule, insult, taunt and deceive all who stand in his way to power. Richardââ¬â¢s skill with language and argument is what enables him to woo Lady Anne, have Clarence thrown in prison and blame the king for Clarenceââ¬â¢s death, all at very little risk to himself. In conclusion, I feel Richard IIIââ¬â¢s unabashed enjoyment of both the power of language and his own villainy makes him a character worthy of both respect and admiration, and therefore I completely agree with this statement. This is because Richardââ¬â¢s unabashed power of language shows off the ingenious wit and intellectual cleverness of the character, actor and playwright, while his own villainy makes the play all the more fascinating and entertaining as his heinous acts become more chilling. How to cite Richard III: Power of Language and Own Villainy, Essay examples
Friday, May 1, 2020
How to promote Nutrition in Dementia Patients- Myassignmenthelp.com
Question: Describe about the Promoting Nutrition in Dementia Patients for A Literature Review. Answer: Introduction Dementia is a mental condition that considerably hinders the daily life of individuals. This clinical condition is manifested by the complications like impaired memory, communication problem, inattentiveness, disturbance in visual perception and reasoning. Dementia develops with mental deterioration and factors like diet and exercise extend considerable influence on the progression of this disease among the affected individuals. Elderly people are the greatest victims of dementia and their mental condition deteriorates due to nutritional inadequacies that require the earliest mitigation for normalizing their lives across the community environment. This literature review explores the nutritional causes of dementia and identifies the evidence-based dietary interventions for effectively treating the clinical manifestations of this complex mental condition. The requirement of enhancing the nutritional status of the demented patients emphasized throughout the length of the literature revi ew in the context of improving their quality of life and mental health outcomes. The literature review effectively explores the pattern of relationship between the quality of life of demented patients and their nutritional requirements in the context of formulating appropriate dietary interventions for mitigating the intensity of their mental symptoms. The focus of this literature review attributes to the exploration of nutritional strategies while including various macronutrients and micronutrients in the diet of the demented patients for reducing the intensity of their cognitive decline. The literature review further explores the therapeutic benefits of vitamins E, B and C and other antioxidants in the context of improving the mental health outcomes of the individuals affected with the pattern of dementia. The literature review focuses on the requirement of developing behavioural and feeding interventions for the demented patients in reducing their feeding dependency and associate d pattern of undernutrition and weight loss experienced by them across the community environment. The literature review also emphasizes the requirement of the effective modification of the mealtime environment of the elderly individuals in the context of reducing their predisposition towards the development of dementia and its associated mental manifestations. Literature Search Strategy This literature search utilized CINAHL database because of its comprehensive attributes and wide utilization by the research community. The search term nutrition in dementia generated 24, 567 results and therefore, Boolean settings were utilized for incorporating multiple keywords like Dementia prevention, nutritional causes of dementia and dementia prophylaxis while searching for the relevant peer reviewed articles matching with the subject of study. Filters were applied to effectively segregate the primary research articles with the secondary ones in the context of their inclusion in the literature review. Indeed, 34 research studies qualified the requirements of the subject of interest in accordance with the credibility findings, bias, confounding factors and study theme. NMC code of conduct and Anglia Ruskin University portal was utilized in searching the evidence based content matching with the theme of the study. Dementia - The Nutritional Context The research findings by (Rossor et al., 2010) indicate dementia as a major public health concern that affects individuals with their age advancement. The pattern of dementia adversely affects the cognitive as well as occupational functioning of the affected patients. This disease influences the patients of age range 30 65 years; however, the prevalence of the disease recorded among the elderly individuals. The findings by (Swaminathan Jicha, 2014) indicate the sustained deficiency of caprylic acid as one of the several causes of the development of Alzheimers dementia in aged people. Therefore, treatment strategies for dementia patients require the administration of medical food and dietary regimen for accomplishing their nutritional deficits in the context of mitigating the symptoms of memory, reasoning, judgment and communication impairment (Swaminathan Jicha, 2014). Evidence-based research literature advocates the high predisposition of the obese and malnourished individuals in terms of developing the patterns of Alzheimers dementia and associated psychosocial manifestations (Hu et al., 2013). Memory loss among the demented patients might occur due to thiamine deficiency arising from malnutrition (Ghosh, 2010). The deficiency of vitamin B 12 in co-morbid states like celiac disease and pernicious anemia leads to the development of neuropsychiatric symptoms that deteriorate the mental health status of the demented patients (Ghosh, 2010). Research findings by (Thaipisuttikul Galvin, 2012) indicate the attribution of metabolic deficiencies in the causation of Alzheimers dementia among the predisposed patients. These deficiencies arise because of dietary mismanagement and nutritional abnormalities among the affected patients. Nutritional inappropriateness also contributes to the development of memory decline and communication deficits among demented patients (Copped et al., 2012). Nutritional deficiencies during the early age facilitate the age related degen eration of human brain. Therefore, the sustained nutritional deficits among the elderly individuals increase their predisposition towards the development of dementia and its associated manifestations. Evidence based research literature reveals the elevated risk of malnutrition among the patients affected with senile dementia and associated psychosocial manifestations (Volkert et al., 2015). This nutritional decline adversely influences the cognitive ability of elderly patients that deteriorates further due to the inappropriate dietary patterns and nutritional management strategies. The sustained deficits of micronutrients and vitamins C and E increase the oxidative stress in the brain that resultantly deteriorates the cognitive, learning and communication abilities of the demented patients (Santos et al., 2014). The research findings by (Polidori Schulz, 2014) indicate the attribution of a defective antioxidant defense mechanism in the causation of dementia among individuals of va rious age groups. Various dietary vitamins and minerals are responsible for maintaining the antioxidant defense mechanism in the human body and therefore their sustained deficiency considerably increases the predisposition of individuals in terms of developing dementia and its associated clinical manifestations. The deficiency in -carotene also deteriorates the cognitive performance and extends detrimental effects on the pattern of dementia among the affected individuals (Polidori Schulz, 2014). The inappropriate administration of the selective serotonin uptake inhibitors inhibits the calcium uptake that resultantly elevates the risk of the individuals in terms of developing mental health problem (Rao et al., 2008)s. Similarly, the deficiency of chromium leads to the development of mental health issues in the affected individuals. The deficiencies of the nutrients like iodine, iron, lithium, selenium and zinc also promote the establishment of mental complications like dementia and other psychosocial conditions (Rao et al., 2008). The findings by (Gu Scarmeas, 2011) indicate the lack of consumption of fruits, vegetables, legumes and nuts as the significant cause of the development of dementia and associated cognitive deficit. The evidence-based analysis by (Ramesh et al., 2010) reveals the high intake of alcohol and saturated fatty acids as one of the important causes of dementia and its cognitive manifestations. Similarly, the sustained deficiency of the methionine-rich proteins promotes the development of dementia and age associated learning and cognitive disabilities among the affected individuals. The adverse eating behavior of the demented individuals creates nutritional imbalance that further deteriorate their cognitive and mental conditions (Ramesh et al., 2010). The clinical findings in (Wald et al., 2011) indicate the attribution of the high levels of homocysteine in the establishment of dementia patterns among the elderly population. The elevated le vel of folate and vitamin 12 along the with the high level of homocysteine increase the risk of the elderly patients in terms of developing Dementia and associated communication deficits (University_of_Oxford, 2007). Dementia Prevention through Nutrition Promotion The research findings by (Canevelli et al., 2016) reveal the potential advantages of various dietary interventions in terms of elevating the neuropsychological test scores of the demented patients. The Mediterranean diet including the mixed nuts and virgin olive oil assists the demented people in terms of enhancing the level of their cognition and memory (Canevelli et al., 2016). The inclusion of antioxidants in the diet of elderly individuals facilitates the reduction in inflammation that reciprocally reduces the scope of dementia establishment among the predisposed people (Middleton Yaffe, 2009). The administration of polyunsaturated fatty acids through vegetables, fruits and fish decreases the risk of individuals of various age groups in terms of developing dementia and associated cognitive decline (Middleton Yaffe, 2009). Research findings by (Dacks et al., 2014) advocate the significance of the pattern of daily walking and administration of the Mediterranean diet in terms of f acilitating loss of weight among demented patients affected with obesity. Indeed, the administration of these integrated techniques assists in improving the quality of life as well as mental health outcomes of the demented patients. The evidence-based findings by (Eskelinen Kivipelto, 2010) advocate the likelihood of the beneficial effects of caffeine administration on the mental health of individuals. Antioxidant capacity and other potential health advantages of caffeine require further exploration in the context of developing prophylactic interventions for reducing the establishment of dementia (Eskelinen Kivipelto, 2010). Dementia in elderly patients is manifested by the accumulation of amyloid and tangles in the necrotic region of the human brain. The modification in the dietary habits of demented patients assists in reversing this abnormal accumulation leading to their subsequent reduction in cognitive decline and memory deterioration (Solomon et al., 2014). Evidence based r esearch literature describes coronary artery disease, diabetes mellitus, lipid problems and arteriosclerosis as the significant causes of vascular dementia among the elderly people (McVeigh Passmore, 2006). Research findings by (Asif, 2014) advocate the requirement of administering cereals, nuts and fruits for enhancing the accumulation of dietary fibers and polyphenols that prevent the pattern of weight gain and assists in controlling the level of blood glucose in the diabetic patients. The reduction in blood glucose level resultantly reduces the predisposition of diabetic patients towards developing dementia and its associated mental health issues. The clinical findings in (Solfrizzi et al., 2011) reveal the protective effect of milk and dairy products in preventing the onset and establishment of dementia manifestations among the predisposed individuals. The evidence-based findings by (Wu et al., 2015) indicate the attribution of omega 3 fatty acids in the causation of dementia among the elderly individuals. Therefore, increased intake of omega 3 fatty acids through diet decreases the likelihood of aged people in terms of acquiring dementia and its clinical complications. Evidence-based findings by (Copped et al., 2012) indicate the positive implications of the concomitant administration of lifestyle and nutritional interventions on the age related cognitive impairment of the individuals. Systematization of dietary intake with the administration of exercise interventions helps in reducing the stress of individuals that eventually decrease the scope of over-nutrition, reduction in physical activity and the establishment of metabolic complications that enhance the likelihood of dementia establishment. The findings by (Copped et al., 2012) also indicate the high level of plasma triglyceride and reduced level of HDL among the demented individuals affected with blood-brain barrier impairment (BBBI). Therefore, the enhancement of HDL component in the diet of th ese patients assists in regularizing their lipid metabolism that significantly influences their mental health outcomes. The research findings by (Hanson et al., 2011) advocate the requirement of administration of oral feeding in demented patients in the context of improving their overall health status. The nurse professionals require the implementation of the NMC code of conduct during the administration of oral feed and should uphold the dignity of the demented patients while extending nutritional management for their health improvement (NMC, 2015). Assisted feeding through intubation might prove painful for the demented patients and predispose them towards the development of gastrointestinal complications. Therefore, the nurse professionals must practice necessary protective measures while administering nutritional support and assistance to the demented patients across the clinical setting. The concomitant administration of multinutrient supplementation and strategies for improvin g the quality of sleep of individuals extends protective effects on their brain that reduces their likelihood of acquiring dementia across the community environment (Yaffe Hoang, 2013). Dementia and Undernutrition/Weight Loss Mechanisms The research findings by (Meijers et al., 2014) indicate the high prevalence of malnutrition among the dementia affected patients. Although, the pattern of weight loss advances with age progression; however, the establishment of dementia facilitates the weight loss mechanisms among the affected patients that prove to be the potential barriers in their care and treatment. Evidence-based research literature advocates the requirement of nutritional intervention for Alzheimer's disease patients who experience the weight loss of more than 5% within tenure of 3 6 months (Droogsma et al., 2015). Furthermore, nutritional assessment proves to be an important tool in determining the undernourished status of demented patients in the context of initiating nutritional management strategies for effectively improving their wellness outcomes. The clinical findings in (Pilleron et al., 2015) reveal the close association of the pattern of cognitive decline in undernutrition status of the demented ind ividuals. The pattern of weight loss caused due to undernutrition reciprocally increases with the dementia stages. This confirms that the severity of dementia manifestations facilitates the progression of weight loss and associated health complications among the affected patients. The pattern of poor nutrition along with alcohol use leads to sustained thiamine deficiency among the demented individuals (Ridley et al., 2013). This thiamine deficiency facilitates the development of cerebellar dysfunction, oculomotor disorders and altered mental status manifested by the pattern of hemorrhagic lesions and neuronal loss during the course of dementia progression. The clinical findings by (Wu Lin, 2015) indicate the potential deterioration in the mealtime performance of the patients in their late stages of dementia. Defective mealtime performance results in nutritional deficiencies among the affected patients that further leads to the progression of their clinical complications. This indic ates the requirement of undertaking memory-training interventions for improving the eating capacity of demented patients. Indeed, improved eating capacity assists the demented patients in terms of overcoming the state of their undernutrition and associated adverse health outcomes. The research findings by (Chow et al., 2009) indicate the pattern of apathy experienced by the demented individuals across the community environment. This apathy results in the development of anorexia and disinterest in feeding leading to undernutrition that adversely influences the cognitive abilities of the affected individuals. The evidence-based findings by (Yildiz et al., 2015) indicate the episodes of hallucinations, delusions, agitation, insomnia, incontinence and immobility experienced by the demented patients under the influence of sustained undernutrition. These adverse episodes further deteriorate the feeding behavior of the affected patients that extends detrimental effects on their metabolic f unctioning and mental health. Strategies for Nutritional Enhancement of Dementia Patients The research findings by (Salv et al., 2011) reveal the reduction in malnutrition risk of the demented patients following the administration of dietary and exercise interventions by trained nutritionists. Research subjects experienced the enhancement of autonomy and nutrition status and reduction in caregiver burden under the influence of the administered dietary approaches. The study by (Bunn et al., 2016) attempted to explore the effects of the interventions like family-style meals, improved the dining room, shared mealtime and enhanced in lightening and aroma of the dining rooms in elevating the nutritional status of the demented patients. The findings of the research study did not reveal concrete evidence regarding the advantages of administered environmental modifications in terms of improving the dietary pattern and nutritional state of the treated patients. However, prospective exploration of strategies highly warranted in the context of understanding their potential for impro ving the nutritional condition of the patients affected with dementia and its adverse mental manifestations. The randomized controlled trial by (Ngandu et al., 2015) attempted to investigate the advantages of the multidomain approaches attributing to cognitive training, dietary interventions, vascular risk monitoring and exercise strategies in terms of preventing the risk of cognitive decline and dementia among elderly patients. The findings of the study revealed the potential of these multidomain strategies for improving the cognitive functioning of the elderly individuals. This also confirms the effective contribution of dietary strategies for improving the mental health of the elderly patients who experience the risk of developing dementia across the community environment. However, prospective studies require execution in the context of exploring the potential of singly administered dietary interventions in improving the mental health status of the demented patients. Conclusion The findings of this literature research evidentially reveal the dietary factors that considerably elevate or decrease the predisposition of individuals towards the development of dementia and its associated mental manifestations. Important dietary constituents like vitamin B complex, vitamins E and C and omega 3 polyunsaturated fatty acids offer a protective role for the elderly people who experience high risk of developing the pattern of dementia across the community environment. The findings also advocate the requirement of implementing various lifestyle modification strategies including exercise interventions and dietary management approaches for stabilizing the metabolic condition of individuals affected with dementia symptoms. The nurse professionals and other members of the healthcare teams must administer evidence-based feeding interventions to maintain the nutritional status of demented patients in the context of treating their symptoms of memory impairment and cognitive de cline. Undernutrition among demented patients is a significant health concern requiring earliest intervention by nutritionists, physicians and nurses in the context of improving their health and wellness across the community environment. Healthcare professionals need to identify the attribution of the sustained nutritional deficits in the causation of dementia and its clinical manifestations among the predisposed individuals. The role of antioxidant defence mechanisms in improving the mental health status of individuals emphasized throughout the course of literature review. Furthermore, the dietary factors that adversely influence the mental health outcomes of demented patients emphasized and appropriate dietary interventions recommended accordingly for the effective enhancement of their cognition, learning and memory. The weight management as well as hypertension and hypercholesterolemia control strategies through nutritional interventions warranted in the context of decreasing the likelihood of elderly patients affected with BBBI towards the acquisition of dementia and its psychosocial manifestations. Indeed, the nutritional management strategies prove to be the significant tools in improving the mental health of the demented patients; however, the prospective research studies warranted for streamlining the integrated nutritional as well as pharmacotherapeutic approaches for reducing the burden of dementia across the community environment. References Asif, M., 2014. The prevention and control the type-2 diabetes by changing lifestyle and dietary pattern. Journal of Education and Health Promotion, 3(1), Available at: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3977406/. Bunn, D.K. et al., 2016. Effectiveness of interventions to indirectly support food and drink intake in people with dementia: Eating and Drinking Well IN dementiA (EDWINA) systematic review. BMC Geriatrics, 16(89), Available at: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4855348/. Canevelli, M. et al., 2016. Nutrition and Dementia: Evidence for Preventive Approaches? Nutrients, 8(3), p.144. Available at: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4808873/. Chow, T.W. et al., 2009. Apathy Symptom Profile and Behavioral Associations in Frontotemporal Dementia vs. Alzheimer's Disease. Archives of Neurology, 66(7), pp.888-93. Available at: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2875777/. Copped, F., Bosco, P., Fuso, A. Troen, A.M., 2012. Nutrition and Dementia. Current Gerontology and Geriatrics Research. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3385589/. Dacks, P.A. et al., 2014. Dementia Prevention: optimizing the use of observational data for personal, clinical, and public health decision-making. The Journal of Prevention of Alzheimer's Disease, 1(2), pp.117-23. Available at: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4487813/. Droogsma, E., Van, A.D. De, D.P.P., 2015. Weight loss and undernutrition in community-dwelling patients with Alzheimer's dementia: From population based studies to clinical management. Zeitschrift fr Gerontologie und Geriatrie, 48(4), pp.318-24. Available at: https://www.ncbi.nlm.nih.gov/pubmed/25990006. Eskelinen, M.H. Kivipelto, M., 2010. Caffeine as a protective factor in dementia and Alzheimer's disease. Journal of Alzheimer's Disease, pp.S167-74. Eskelinen, M.H. Kivipelto, M., 2010. Caffeine as a protective factor in dementia and Alzheimer's disease. Journal of Alzheimer's Disease, pp.S167-74. Available at: https://www.ncbi.nlm.nih.gov/pubmed/20182054. Ghosh, A., 2010. Endocrine, metabolic, nutritional, and toxic disorders leading to dementia. Annals of Indian Academy of Neurology, 13(2), pp.S6368. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3039161/. Gu, Y. Scarmeas, N., 2011. Dietary Patterns in Alzheimers Disease and Cognitive Aging. Current Alzheimer Research Journal, 8(5), pp.510-19. Available at: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3283139/. Hanson, L.C., Ersek, M., Gilliam, R. Carey, T.S., 2011. Oral Feeding Options for Patients with Dementia: A Systematic Review. Journal of the American Geriatrics Society, 59(3), pp.463-72. Available at: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3164780/. Hu, N. et al., 2013. Nutrition and the Risk of Alzheimer's Disease. BioMed Research International. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3705810/. McVeigh, C. Passmore, P., 2006. Vascular dementia: prevention and treatment. Clinical Interventions in Aging, 1(3), pp.229-35. Available at: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2695177/. Meijers, J.M., Schols, J.M. Halfens, R.J., 2014. Malnutrition in care home residents with dementia. The Journal of Nutrition, Health and Aging, 18(6), pp.595-600. Available at: https://www.ncbi.nlm.nih.gov/pubmed/24950150. Middleton, L. Yaffe, K., 2009. PROMISING STRATEGIES FOR THE PREVENTION OF DEMENTIA. Archives of neurology, 66(10), pp.1210-15. Available at: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2762111/. Ngandu, T. et al., 2015. A 2 year multidomain intervention of diet, exercise, cognitive training, and vascular risk monitoring versus control to prevent cognitive decline in at-risk elderly people (FINGER): a randomised controlled trial. Lancet, pp.2255-63. Available at: https://www.ncbi.nlm.nih.gov/pubmed/25771249. NMC, 2015. The Code for nurses and midwives. [Online] Available at: https://www.nmc.org.uk/standards/code/ [Accessed 29 October 2016]. Pilleron, S. et al., 2015. Association between mild cognitive impairment and dementia and undernutrition among elderly people in Central Africa: some results from the EPIDEMCA (Epidemiology of Dementia in Central Africa) programme. The British Journal of Nutrition, 114(2), pp.306-15. Available at: https://www.ncbi.nlm.nih.gov/pubmed/26099336. Polidori, M.C. Schulz, R.J., 2014. Nutritional contributions to dementia prevention: main issues on antioxidant micronutrients. Genes Nutrition, 9(2), p.382. Available at: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3968291/. Ramesh, B.N. et al., 2010. Neuronutrition and Alzheimer's Disease. Journal of Alzheimer's Disease, 19(4), pp.1123-39. Available at: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2931824/. Rao, T.S.S., Asha, M.R., Ramesh, B.N. Rao, K.S.J., 2008. Understanding nutrition, depression and mental illnesses. Indian Journal of Psychiatry, 50(2), pp.77-82. Available at: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2738337/. Ridley, N.J., Draper, B. Withall, A., 2013. Alcohol-related dementia: an update of the evidence. Alzheimer's Research Therapy, 5(1), Available at: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3580328/. Rossor, M.N. et al., 2010. The diagnosis of young-onset dementia. The Lancet Neurology, 9(8), pp.793-806. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2947856/. Salv, A. et al., 2011. Health and nutrition promotion program for patients with dementia (NutriAlz): cluster randomized trial. The Journal of Nutrition, Health and Aging, 15(10), pp.822-30. Available at: https://www.ncbi.nlm.nih.gov/pubmed/22159768. Santos, J.R., Gois, A.M., Mendona, D.M.F. Freire, M.A.M., 2014. Nutritional status, oxidative stress and dementia: the role of selenium in Alzheimer's disease. Frontiers in Aging Neuroscience, 6(206). 10.3389/fnagi.2014.00206. Solfrizzi, V. et al., 2011. Diet and Alzheimer's disease risk factors or prevention: the current evidence. Expert Review of Neurotherapeutics, 11(5), pp.677-708. Solomon, A. et al., 2014. Advances in the prevention of Alzheimers disease and dementia. Journal of Internal Medicine, 275(3), pp.229-50. Available at: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4390027/. Swaminathan, A. Jicha, G.A., 2014. Nutrition and prevention of Alzheimers dementia. Frontiers in Aging Neuroscience, 6(282). https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4202787/. Thaipisuttikul, P. Galvin, J.E., 2012. Use of medical foods and nutritional approaches in the treatment of Alzheimers disease. Clinical Practice, 9(2), pp.199-209. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3556480/. University_of_Oxford, 2007. Homocysteine, B vitamins, and the risk of dementia. The American Journal of Clinical Nutrition, Available at: https://ajcn.nutrition.org/content/85/2/329.full. Volkert, D. et al., 2015. ESPEN guidelines on nutrition in dementia. Clinical Nutrition, 34(6), pp.1052-73. https://www.ncbi.nlm.nih.gov/pubmed/26522922. Wald, D.S., Kasturiratne, A. Simmonds, M., 2011. Serum homocysteine and dementia: meta-analysis of eight cohort studies including 8669 participants. Alzheimer's and Dementia, 7(4), pp.412-17. Available at: https://www.ncbi.nlm.nih.gov/pubmed/21784352. Wu, S. et al., 2015. Omega-3 fatty acids intake and risks of dementia and Alzheimer's disease: a meta-analysis. Neuroscience and Behavioural Reviews, pp.1-9. Available at: https://www.ncbi.nlm.nih.gov/pubmed/25446949. Wu, H.S. Lin, L.C., 2015. Comparing cognition, mealtime performance, and nutritional status in people with dementia with or without ideational apraxia. Biological Research for Nursing, 17(2), pp.199-206. Available at: https://www.ncbi.nlm.nih.gov/pubmed/24924352. Yaffe, K. Hoang, T., 2013. Nonpharmacologic treatment and prevention strategies for dementia. Continuum Minneapolis, Minn, 19(2), pp.372-81. Available at: https://www.ncbi.nlm.nih.gov/pubmed/23558483. Yildiz, D. et al., 2015. Malnutrition is associated with dementia severity and geriatric syndromes in patients with Alzheimer disease. Turkish Journal of Medical Sciences, 45(5), pp.1078-81. Available at: https://www.ncbi.nlm.nih.gov/pubmed/26738350.
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